So they start grabbing the calcium that's floating around, causing calcium to unbind from the troponin.

所以它们开始抓住漂浮在周围钙，导致钙与肌钙蛋白解绑。

The amount of troponin released is proportional to the infarct size.

肌钙蛋白释放量与梗死面积成正比。

Namely, actin is blocked by a couple of protein bodyguards — called tropomyosin and troponin — which keep getting in the way.

也就是说，肌动蛋白会被蛋白镖阻断——原肌球蛋白和肌钙蛋白——它们一直碍事。

As myocardial muscle dies, it releases cardiac markers such as cardiac troponin into the blood stream.

当心肌死亡时，它向血流中释放心肌肌钙蛋白等心脏标。

Cardiac troponins are the markers of choice as they become elevated early in the disease process.

心肌肌钙蛋白是选择标志，因为它们在疾病过程早期就升高。

He says it's hard to draw conclusions about elevated troponin levels since normal hearts can have elevated troponin levels, too.

他说很难就肌钙蛋白水平升高下结论， 因为正常心脏肌钙蛋白水平也会升高。

Most patients with infarction show elevated troponin levels within 6 hours.

大多数脑梗死患者在6小时肌钙蛋白水平升高。

That's because the protein troponin just loves to bind with calcium, and remember: When stuff binds to proteins, the proteins change shape.

这是因为肌钙蛋白太喜欢与钙结合了，住：当与蛋白结合时，蛋白会改变形状。

They found that 78 had signs of heart damage, including elevated levels of troponin, proteins released by the heart muscle when it is damaged.

他们发现 78 人有心脏受损迹象，包括肌钙蛋白水平升高， 心肌受损时会释放蛋白。

Now research carried out in Scotland and United States has led to the development of a much more sensitive troponin test that means many more patients could be quickly reassured their chest pain is nothing to worry about.

现在苏格兰和美国进行一项研究让肌钙蛋白检测方法更加成熟，这意味着可以让更多病人确认自己胸痛并不是心脏病发。

Immediately after a muscle fiber is stimulated by a nerve — when calcium ions are flooding into the sarcomeres to pull away those two protein bodyguards of tropomyosin and troponin from the actin — that's called the latent period.

肌肉纤维被神经刺激后立即——钙离子涌入肌节，将肌动蛋白中护原肌球蛋白和肌钙蛋白蛋白带走——这称为潜伏期。

So the calcium latches on to the troponin and causes it to pull the other bodyguard protein — the tropomyosin — away from the sites on the actin strands that the myosin really wants to get its paws on.

所以钙锁住肌钙蛋白，并使其将另一种护蛋白——原肌球蛋白——从肌球蛋白真正想要抓住肌动蛋白链上位置拉走。