Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目的观察尼麦角林治疗多发性死性痴呆临床疗效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

颈动脉疾病导认知障碍可能与低灌注、白质病变、无症状死、腔隙性死和神经元变性有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大部分被旧灶液化坏死后留下的高信号脑脊液。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

性粒细胞广泛浸润，核溶解、胞浆减少，是典型的急性心肌死的1-4天的表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录心率、血压、左心室内压、左心室内压微分变化。同时观察银杏黄酮对家兔急性心肌血／再灌注治疗后减轻心肌死的情况。

Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目观察尼麦角林治疗多发性梗性痴呆临床疗效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

颈动脉疾病导认知障碍可能与低灌注、白质病变、无症状梗、腔隙性梗和神经元变性有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大部分被旧梗塞灶液化坏后留下高信号脑脊液替代。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

中性粒细胞广泛浸润，核溶解、胞浆减少，是典型急性心肌梗1-4表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录心率、血压、左心室内压、左心室内压微分变化。同时观察银杏黄酮对家兔急性心肌缺血／再灌注治疗后减轻心肌梗情况。

Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目观察尼麦角林治疗多发性梗性痴呆临床疗效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

颈动脉疾病导认知障碍可能与低灌注、白质病变、无症状梗、腔隙性梗和神经元变性有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大部分被旧梗塞灶液化留下高信号脑脊液替代。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

中性粒细胞广泛浸润，核溶解、胞浆减少，是典型急性心肌梗1-4天表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录心率、血压、左心室内压、左心室内压微分变化。同时观察银杏黄酮对家兔急性心肌缺血／再灌注治疗减轻心肌梗情况。

Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目的观察尼麦角林治疗多发性梗死性痴呆临床疗效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

疾病导认知障与低灌注、白质病变、无症状梗死、腔隙性梗死和神经元变性有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大部分被旧梗塞灶液化坏死后留下的高信号脑脊液替代。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

中性粒细胞广泛浸润，核溶解、胞浆减少，是典型的急性心肌梗死的1-4天的表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录心率、血压、左心室内压、左心室内压微分变化。同时观察银杏黄酮对家兔急性心肌缺血／再灌注治疗后减轻心肌梗死的情况。

Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目的观察尼麦角林治疗多发性梗死性痴呆临床疗效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

颈动脉疾病导认知障碍可能与低灌注、白质病变、无症状梗死、腔隙性梗死和神经元变性有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大分被旧梗塞灶液化坏死后留下的脑脊液替代。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

中性粒细胞广泛浸润，核溶解、胞浆减少，是典型的急性心肌梗死的1-4天的表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录心率、血压、左心室内压、左心室内压微分变化。同时观察银杏黄酮对家兔急性心肌血／再灌注治疗后减轻心肌梗死的情况。

Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目的观察尼麦角多发性梗性痴呆临床效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

颈动脉疾病导认知障碍可能与低灌注、白质病变、无症状梗、性梗和神经元变性有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大部分被旧梗塞灶液化坏后留下的高信号脑脊液替代。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

中性粒细胞广泛浸润，核溶解、胞浆减少，是典型的急性心肌梗的1-4天的表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录心率、血压、左心室内压、左心室内压微分变化。同时观察银杏黄酮对家兔急性心肌缺血／再灌注后减轻心肌梗的情况。

Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目的观察尼麦角林治疗多发梗死痴呆临床疗效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

颈动脉疾病导认知障碍可能与低灌注、白质病变、无症状梗死、腔隙梗死和神经元变有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶脑软化额极大部分被旧梗塞灶液化坏死后留下的高信号脑脊液替代。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

中粒细胞广泛浸润，核溶解、胞浆减少，是典型的急梗死的1-4天的表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录率、血压、左室内压、左室内压微分变化。同时观察银杏黄酮对家兔急缺血／再灌注治疗后减轻梗死的情况。

Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目的观察尼麦角林治疗多发性梗死性痴呆临床疗效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

颈疾病导认知障碍可能与低灌注、白质病变、无症状梗死、腔隙性梗死和神经元变性有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右叶脑软化部分被旧梗塞灶液化坏死后留下的高信号脑脊液替代。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

中性粒细胞广泛浸润，核溶解、胞浆减少，是典型的急性心肌梗死的1-4天的表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录心率、血压、左心室内压、左心室内压微分变化。同时观察银杏黄酮对家兔急性心肌缺血／再灌注治疗后减轻心肌梗死的情况。

Objective To observe the effect on nicergoline in treatment of multi infarct dementia.

目观察尼麦角林治疗多发性死性痴呆临床疗效。

The mechanisms of cognitive impairment caused by carotid diseases are associated with hypoperfusion, leukodystrophy, silent or lacunar infarcts and neuronal degeneration.

颈动脉疾病导认知障碍可能与低灌注、白质病变、无症状死、腔隙性死和神经元变性有关。

Right frontal encephalomalacia The frontal pole is largely replaced with the high signal of cerebrospinal fluid left after liquifaction necrosis in this old infarct.

右额叶软化额极大部分被灶液化坏死后留下高信号脊液替代。

The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset.

中性粒细胞广泛浸润，核溶解、胞浆减少，是典型急性心肌死1-4天表现。

We recorded the changes of the heart rate the blood pressure, and the left ventricle pressure, and observed Ginkgetin effect on reducing the myocardial infarct death rate of AMI - reperfused rabbits.

记录心率、血压、左心内压、左心内压微分变化。同时观察银杏黄酮对家兔急性心肌缺血／再灌注治疗后减轻心肌死情况。